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anti lc3i ii  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc anti lc3i ii
    Anti Lc3i Ii, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 41 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/lc3i+ii/Phospho-IRF-3+(Ser379)+Rabbit+mAb/pmc13004953-288-43-44
    Average 95 stars, based on 41 article reviews
    anti lc3i ii - by Bioz Stars, 2026-09
    95/100 stars

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    Related Articles

    SDS Page:

    Article Title: Philadelphus tenuifolius Leaf Extract Exhibits Anti-Tuberculosis Activity by Enhancing Host Autophagy and Immunity: A Promising Host-Directed Therapeutic Candidate
    Article Snippet: .. Proteins were separated by SDS-PAGE and probed with primary antibodies against ERK1/2, p38, JNK, p-ERK1/2, p-p38, p-JNK, p62 and LC3I/II (Cell Signaling Technology, USA). ..

    Incubation:

    Article Title: 457 nm LED blue light inhibits oral squamous cell carcinoma cell proliferation via the PI3K/AKT pathway and autophagy.
    Article Snippet: Background Photobiomodulation (PBM) therapy, utilizing light to regulate cellular processes, holds promise as a cancer treatment.. We aimed to explore PBM's impact on two oral squamous cell carcinoma (OSCC) cell lines, Cal-27 and SCC-25, under varying blue light irradiation conditions.. Methods Cal-27 and SCC-25 cells were exposed to 457 nm blue light at varying irradiances (2.5–70 mW/cm2) and radiant exposure levels (3–84 J/cm2).

    Article Title: Honokiol modulates invasion and autophagy in pancreatic cancer via the miR-101/QKI axis in cancer-associated fibroblasts
    Article Snippet: .. After blocking with 5 % nonfat milk for 2 h, membranes were incubated with diluted primary antibodies against α-SAM (1:1000, ab109197, abcam), FAP (1:1000, ab314456, abcam), FSP1 (1:1000, ab197896, abcam), GAPDH (1:1000, ab8245, abcam), QKI (1:1000, sc-517305, Santa-Cruz) or LC3I/II (1:1000, 4108, Cell Signaling Technology). ..

    Article Title: Hemp seed extract exerts cytostatic effects through metabolic stress and autophagy modulation in malignant cells.
    Article Snippet: Gels were transferred on PVDF membranes (Bio-Rad Laboratories, Milan, Italy) using a Trans Blot Turbo System (Bio-Rad). .. Membranes were blocked with 5% non fat dry milk in T-TBS 0.1% followed by incubation with the primary antibodies: anti LC3I/II (cat. # 12741S) and p62SQSTM1 (cat. # 5114S), anti pAMPKαThr172 (cat. # 2535S), anti p27kip1 (cat. # 3688) (Cell Signalling Technology, Milan, Italy), anti α-Tubulin was from MERK-Millipore (cat. # T9026) antibodies. .. The immunoblots were performed by means of standard procedures and developed by the ECL Plus Western blot detection system (Cytiva, Euroclone, Milan, Italy).

    Blocking Assay:

    Article Title: Honokiol modulates invasion and autophagy in pancreatic cancer via the miR-101/QKI axis in cancer-associated fibroblasts
    Article Snippet: .. After blocking with 5 % nonfat milk for 2 h, membranes were incubated with diluted primary antibodies against α-SAM (1:1000, ab109197, abcam), FAP (1:1000, ab314456, abcam), FSP1 (1:1000, ab197896, abcam), GAPDH (1:1000, ab8245, abcam), QKI (1:1000, sc-517305, Santa-Cruz) or LC3I/II (1:1000, 4108, Cell Signaling Technology). ..

    Membrane:

    Article Title: Betulinic acid attenuates lipopolysaccharide-induced cardiac injury by promoting mitophagy with enhancing PINK1/Parkin and suppressing BNIP3.
    Article Snippet: Betulinic acid (BA), a natural pentacyclic triterpene, has been shown to promote autophagy and attenuate sepsisinduced organ injury, yet its role in mitophagy-mediated cardioprotection remains unclear.. Here, we evaluated the effects of oral BA treatment (25 mg/kg, 5 days) on lipopolysaccharide (LPS)-induced cardiac injury in male Sprague-Dawley rats.. BA significantly improved cardiac function, reduced myocardial injury markers (cardiac troponin I, creatine kinase-MB), and suppressed inflammatory (tumor necrosis factor-α, interleukin-1β, myeloperoxidase activity) and oxidative responses in LPS-induced sepsis.



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